, .  et al. 0

the high expression of this gene in fetal brain and in the adult cerebral cortex could be consistent with a role in brain development and/or cognitive function.", .  et al. 0

, .  et al. 0

Jacques G., .  et al. 0

Lisa G.; Gunkel

Gene. 1999 Aug 5, .  et al. 0

Villard L

Kammoun F, .  et al. 0

Fakhfakh F

Nancy, .  et al. 0

Cornel; Missirian

E., .  et al. 0

L.; Gecz

, .  et al. 0

126 by PGY-2 residents, .  et al. 0

and 131 by PGY-3 residents. A total of 35% (160 of 449) of intubation procedures were never successful by pediatric hou

Am J Hum Genet. 1999 Aug, .  et al. 0

Lossi AM

Roux JC. "BMC Neurosci. 2011 May 24, .  et al. 0

Lester T, .  et al. 0

Villard L

in a child presenting with ID and an autism spectrum disorder (ASD). The second CNV is a partial deletion of KLHL15, .  et al. 0

in a patient with severe ID

, .  et al. 0

St-Onge J, .  et al. 0

Duffourd Y

, .  et al. 0

Am J Med Genet. 1999 Apr 2, .  et al. 0

Holden JJ

Villard L. "J Neurosci Res. 2010 May 15, .  et al. 0

to a lesser extent, .  et al. 0

in the glutamate pathway. The implication of the GABA pathway in the RTT neuropathology was further confirmed using an in vivo treatment with a GABA reuptake inhibitor that significantly improved the lifespan of Mecp2-deficient mice. Our results confirm that RTT mouse present a deficit in the GABAergic pathway and suggest that GABAergic modulat